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mouse anti havcr kim 1  (R&D Systems)


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    R&D Systems mouse anti havcr kim 1
    Mouse Anti Havcr Kim 1, supplied by R&D Systems, used in various techniques. Bioz Stars score: 96/100, based on 297 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+anti+kim+1+antibody/Mouse+TIM-1%2FKIM-1%2FHAVCR+Antibody/pm41981279-218-9-15
    Average 96 stars, based on 297 article reviews
    mouse anti havcr kim 1 - by Bioz Stars, 2026-10
    96/100 stars

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    Article Title: Human kidney-derived tubular organoid, tubuloid, recapitulates cellular senescence, inflammation and fibrosis by repeated-cisplatin treatment
    Article Snippet: As primary antibodies, rabbit anti-LRP2 / Megalin antibody (1:100) (ab236244, Abcam, MA, USA), rabbit anti-γH2AX antibody (1:200) (#9718, Cell Signaling Technology, MA, USA), mouse anti-Na + /K + -ATPase antibody (1:200) (05-369, Sigma-Aldrich), mouse anti-KIM-1 antibody (1:200) (AF1750, R&D systems, MN, USA), rabbit anti-caspase-3 antibody (1:200) (#9664, Cell Signaling Technology), rabbit anti-Vimentin antibody (1:200) (10366-1-AP, Proteintech, IL, USA), rabbit anti-p16-INK4A polyclonal antibody (1:200) (10883-1-AP, Proteintech), rabbit anti-IL-1β polyclonal antibody (1:200) (bs-0812R, Bioss, MA, USA) and rabbit anti-αSMA antibody conjugated with Cy3 (1:400) (C6198, Sigma-Aldrich) were used.

    Article Title: Human kidney-derived tubular organoid, tubuloid, recapitulates cellular senescence, inflammation and fibrosis by repeated-cisplatin treatment
    Article Snippet: Rabbit anti-p16-INK4A polyclonal antibody (1:200) (Proteintech), mouse anti-KIM-1 antibody (1:200) (R&D systems), rabbit anti-caspase-3 antibody (1:200) (Cell Signaling Technology) and rabbit anti-Vimentin antibody (1:200) (Proteintech) were used as primary antibodies.



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    Macrophage‐Specific Deletion of Zxdb Protects Against Sepsis‐Induced Acute Kidney Injury. (A) Generation strategy for myeloid‐specific Zxdb knockout (Mac‐Zxdb‐KO) mice. (B) H&E staining of kidney sections from Sham, WT SI‐AKI, and Mac‐Zxdb‐KO SI‐AKI mice. (C‐D) Serum creatinine (Scr) (C) and blood urea nitrogen (BUN) (D) levels in Sham, WT SI‐AKI, and Mac‐Zxdb‐KO SI‐AKI mice at 24 h post‐CLP. (E) KIM‐1 immunohistochemistry of kidney sections from Sham, WT SI‐AKI, and Mac‐Zxdb‐KO SI‐AKI mice. (F) TUNEL staining of kidney sections from Sham, WT SI‐AKI, and Mac‐Zxdb‐KO SI‐AKI mice. (G) Immunofluorescence of kidney sections stained for F480, CD86, and CD206. (H) Serum IL‐1β, IL‐10, and TNF‐α levels in Sham, WT SI‐AKI, and Mac‐Zxdb‐KO SI‐AKI mice at 24 h post‐CLP. * p < 0.05, ** p < 0.01, *** p < 0.001.

    Journal: The FASEB Journal

    Article Title: ZXDB Drives Macrophage Inflammatory Programming in Sepsis‐Induced Acute Kidney Injury by Recruiting EIF4A3 to Enhance ACACA Translation

    doi: 10.1096/fj.202502962RR

    Figure Lengend Snippet: Macrophage‐Specific Deletion of Zxdb Protects Against Sepsis‐Induced Acute Kidney Injury. (A) Generation strategy for myeloid‐specific Zxdb knockout (Mac‐Zxdb‐KO) mice. (B) H&E staining of kidney sections from Sham, WT SI‐AKI, and Mac‐Zxdb‐KO SI‐AKI mice. (C‐D) Serum creatinine (Scr) (C) and blood urea nitrogen (BUN) (D) levels in Sham, WT SI‐AKI, and Mac‐Zxdb‐KO SI‐AKI mice at 24 h post‐CLP. (E) KIM‐1 immunohistochemistry of kidney sections from Sham, WT SI‐AKI, and Mac‐Zxdb‐KO SI‐AKI mice. (F) TUNEL staining of kidney sections from Sham, WT SI‐AKI, and Mac‐Zxdb‐KO SI‐AKI mice. (G) Immunofluorescence of kidney sections stained for F480, CD86, and CD206. (H) Serum IL‐1β, IL‐10, and TNF‐α levels in Sham, WT SI‐AKI, and Mac‐Zxdb‐KO SI‐AKI mice at 24 h post‐CLP. * p < 0.05, ** p < 0.01, *** p < 0.001.

    Article Snippet: Sections were blocked and incubated overnight at 4°C using primary antibody against KIM‐1 (AF1817; R&D Systems, 1:200).

    Techniques: Knock-Out, Staining, Immunohistochemistry, TUNEL Assay, Immunofluorescence